For breath easier.

 Am used fluorography testing and pneumosclerosis diagnosed in me, doctor shared about an adult disease is, cure not prescribed. Asking help in zoz  specialists.

Am on pension, more ,than 14 years sufferings from pneumosclerosis in roots of left lung, which remedies would help me.

 Diseases for breath organs are   knowing everybody, but theirs are seem are not angry.

But these diseases are starting in organism dangerous process is calling pneumosclerosis, so is calling replacing a normal tissue of lungs a connective tissue.

 A saturating blood by oxygen happening in lung bladders are alveoles, in pneumosclerosis slowly as by measure a growing a scar /connective/ tissue are more pressing small blood vessels are capillaries, thus destroying for alveoles feeding ,they are dead. Decreasing breathe surface for lungs, gas exchange destroying, you are often breathing, that in lungs entering less oxygen  ,than a needing for organism. Alveoles remaining are working with a high loading, taking oneself more air, than usual. They are stretching in result, loosing elastic and not a full collapsing in breathe out, power for breathe out decreasing and his continuation increasing ,thus a not rare emphysema displaying, as for pneumosclerosis accompanies, but pnemosclerosis hardening a course for diseases. Pneumosclerosis reason are not cured till end lungs inflammations, obstructive lung disease, tuberculosis, pleuritis,  sarcoidosis, mycosis ,pneumoconiosis, Wegner s granulomatous, atelectasis, and in children's pneumosclerosis displaying after measles or whooping cough, if disease having in result of chronic obstructive lungs diseases ,thus in increasing connective tissue are diagnosing emphysema.

 To pneumosclerosis leading  are genetical systemic diseases as systemic lupus erythematous, scleroderma, systemic vasculitis, entering foreign items in bronchus, damaging lungs and breast cells , sometime disease development tie with thromboembolic of lung artery and cardiovascular work wrong, a long breathing dust, or toxic substances in air, long smoking, ionized radiant .

These are diffusion as spreading  and nidus as local pneumosclerosis , last is big and small nidus. And in depending on from growing size a connective tissue are distinguishes fibrosis, sclerosis, cirrhosis lungs. In pneumosclerosis scar tissues changes in lungs are moderated having, and in pneumosclerosis happening a most full replacement alveoles and a part bronchus and vessels of connective tissue.

 And pneumosclerosis finding by chance in planing x-ray testing , as on the professional checking, or visiting medical place as by other disease complain.

 And till a time, patient for a not feeling changing, and health is ok. As after big loading , in fast walk, or on steps raising you are feeling dyspnoe. And patient thinks about its happening in adults or in cardiovascular failure.  Clinical symptomatic for pneumosclerosis  is depending on from the level a spreading process in lungs, If damage for parts lungs is not big, thus a nothing displaying in diseases and medical help is not needing.If damage for part is expressed,thus symptoms are dyspnoe, attacks with a cough, dry, tiredmess, dizziness.Thus a disease a not self, thus symptoms are same with disease, which had need a reason for pneumosclerosis formed.