Hard correlation.

 As since my childhood am having hard nodules on my body after injures, as parents and me not saw it, but recently am entered in emergency room as with asphyxia attack, and thee two processes are correlations. And diagnosis put me as inborn form of angioneurotic edema, and adviced me to check for health, if no, this will be end a tragedy. 

As lots of patients hearing this diagnosis are thinking about a lots of itching spots ,red colours as catching layers for mucus and subcutaneous cellular is an all body answers allergist immunilogist Treskunov.

And a most often this calling edema Quince,as a first german doctor  opened this disease is on 19 century   as a this edema kind is easy diagnosing and easy curing.

And other in inborn type as genetical edema,as rare meeting and harder and not timely diagnosing. As here are edema hurting, hardening, not itching, usual color over edema, but a rish here edema meeting in any point of organism,as in throat ,thus asphyxia reason, and somethimes edema causing an acute coronary failure,as myocarditis infarctus, acute urine delay as of urinary edema or canal, as a staying bowel acute obstruction.And a reason for edema are many, but importance isin defect of a most power immunity weapon is complement system A complement are ferments selection,diluting protein foregns as in blood having,and theirs cascade directing for organism protect against entering in a blood as enemy,as against a harful affect  a system , her function regulates are specifical proteins as inhibitors,they are timely stopping reaction , preventing a damaging are own tissues,and if you are since a born a got defect inhibitor ,thus tissues are stopping keeping protecting and in result they are getting edema hard. As for diagnosis putting a taking clinical picture, level activity for inhibitor C1  and genetical affecting,as  would this edema had in parents? And in diagnosis putting a not right curing is Quince edema with glucocorticosteroids or anti histamins, as curing in neonatal form,they are here not effectivity.As a problem desiding here with entering inhibitor C1  as of fresh frozen plazma  as remedies are berinert, cinrize, and sucess tested by clinicaly a blockage  receptors bradicinin ,which is in fault in edema display, firazir.

And if in absence these remedies use are amine capronic acid, or tranexamic as a most save with a thrombosis complications.

As for a genetical edema use a remedies of male sex testosterone or anabolic steroids, they are stimulating sysnthesis C1  inhibitors for cells a liver, and self curing with these remedies are avoiding!