Chronic polyarthritis.

 By a social point of view this is a most essential from rheumatic diseases . And pathogenesis for this disease is not known, nature his is problematically. And this is a common disease, but not a congestion for local joint diseases, as she often damages are many joints, thus need to suffect, that here we are having before itself as infection factor.And in medic works ethnology has  been not opened. And cases, beginning of acute and under acute process, with a high temperature are displaying is most favourable.And in weak display, without fever as on begin stage , extremities are cold and moisturize. And just not importance on 0.2-0.3 C temperature rising. And swelling in interphalangeal and metatarsophalangeal joints,after are fingers on the hands, legs, wrists, ankle, and later damaging joints for elbows, knees. Joints of hips and shoulders damaging a most latest. And inflammation beginning from a synovial membrane and in the walls of joints  cavities, and often finding intra-articular  exudate as liquid, after ,like jelly.And other form,where is  intra-articular exudate is less pronounced, but all an own layers of joints walls are swelling. After beginning reaction with a serious destroys for mobility this organ. And damaging joints are never limiting, just with cartilages and bones. All are prearticular histologic systems as connective tissue, tensions membrane, muscle elements, nerves, vessels are taking part, as on a big distance too takes part, and in inflammation edema and for a future are joints deformations. And edema squeezes different capillaries nets, exchange  between theirs and extracellular liquids are stopping, and dehydration beginning, as preceding for shortening. As of blood circulation failure for muscles is muscle atrophy displaying.And this is not arthrogen  muscles atrophy, but not a refectory atrophy, this a muscle cappilaropathy, as insufficient supply a blood a lymph, and here is atrophy. And blood supply restoring and atrophy disappearing. We are known, that muscle atrophy removing, if a joint is curing, but why? Because blood supply restored. And why to search explain this muscle atrophy in fog s hypothesis/damage a neuro vegetative system, neuritis for sympatric system and etc, when a having a visible, sense factor, and blood supply failure for capillary net, with her results, as anoxemia, feeding failure, amino acids failure, glucose, electrolytes, enzymes, catalytic ferments, water with a normal Ph, and here are simple, logic and base physiologic trues. A medical mind is need to be a exactly ,ability to give explain, agreed with applied physiology. And so calling neuro vegetative symptoms are feeling   a cold, sweating extremities, skin diseases, and sclerosis these are different displays fir insufficient circulation in pre capillary arterioles and capillaries .And speech here is about blockage capillary circulation, this a capillaropathy, which  you can to see and observe with a capillary  scope and cure with a capillary therapy. As decreasing a muscle, peri neural, intra neural cappilaropathy , regulating structure for  extracellular liquids, chopping by the balneotherapy slags depositing , as in tension staying, improving a feeding for prearticular tissues are leading a back development for rheumatic diseases. And destroy for periphery circulation a blood is plays in a clinical picture for rheumatism a role is more importance,than though till today/Kahlmeter, 1949, page 53./

And in rheumatism pathogenesis  are will not understand nothing, if will not study a good capillaropathy as a base and decisive a pathogenic factor.And results of water warm cure can be to explain just as therapy for capillaries. And why  a use such fantastic therms as cytophylaxin as an effect of oligo mineral elements, stimulating a neuro vegetative system?  As an acute joint rheumatism,and chronic are common diseases.And heart involving and blood system to pathophysiological processes are not  rare phenomenons. And in chronic rheumatism, heart involving just on 40 percent cases. And here are periphery vessels are more taking part in pathologic process,than in a case of acute rheumatism. And  researches by Scandinavian scientists /Faber, Borries, Jansen, Kahlmeter has been established, that in chronic rheumatism is anaemia displaying as very pronounced  with an insufficient serum ferum.

And in chronic rheumatism  as in chronic lung tuberculosis a need to do difference between anatomic and functional restoring a function. And in this a goal here having restoring for functions, but not disappearing deformations  for bones and joints on x-ray pictures.